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anti-mpd1 antibodies clone rmp1-14  (Bio X Cell)

 
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    Bio X Cell anti-mpd1 antibodies clone rmp1-14
    Anti Mpd1 Antibodies Clone Rmp1 14, supplied by Bio X Cell, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/anti-mpd1 antibodies clone rmp1-14/product/Bio X Cell
    Average 90 stars, based on 1 article reviews
    anti-mpd1 antibodies clone rmp1-14 - by Bioz Stars, 2026-02
    90/100 stars

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    Combination of FAK inhibition and <t>anti-PD1</t> therapy effectively inhibited the growth of HCC in mice (A) After 4 weeks of plasmid injection in C57BL/6 J mice, a primary hepatocellular carcinoma model was established and the mice were randomly divided into four groups (Placebo group, n = 7; Anti-PD1 group, n = 7; FAK inhibitor group, n = 7; Combination group, n = 8), and specific information about the administration (time, dosage, and method). (B) The mouse liver after 2 weeks of medication. (C–D) The liver weight of mice and the liver weight/body weight of mice were compared in each group (Placebo group, n = 7; Anti-PD1 group, n = 7; FAK inhibitor group, n = 7; Combination group, n = 8). Significance identification: ns, p ≥ 0.05; *, p < 0.05; **, p < 0.01; ***, p < 0.001. (E) HCC tissues of mice were histologically analyzed by H and E staining (scale bars, 400 μm).
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    Combination of FAK inhibition and anti-PD1 therapy effectively inhibited the growth of HCC in mice (A) After 4 weeks of plasmid injection in C57BL/6 J mice, a primary hepatocellular carcinoma model was established and the mice were randomly divided into four groups (Placebo group, n = 7; Anti-PD1 group, n = 7; FAK inhibitor group, n = 7; Combination group, n = 8), and specific information about the administration (time, dosage, and method). (B) The mouse liver after 2 weeks of medication. (C–D) The liver weight of mice and the liver weight/body weight of mice were compared in each group (Placebo group, n = 7; Anti-PD1 group, n = 7; FAK inhibitor group, n = 7; Combination group, n = 8). Significance identification: ns, p ≥ 0.05; *, p < 0.05; **, p < 0.01; ***, p < 0.001. (E) HCC tissues of mice were histologically analyzed by H and E staining (scale bars, 400 μm).

    Journal: Frontiers in Pharmacology

    Article Title: A FAK Inhibitor Boosts Anti-PD1 Immunotherapy in a Hepatocellular Carcinoma Mouse Model

    doi: 10.3389/fphar.2021.820446

    Figure Lengend Snippet: Combination of FAK inhibition and anti-PD1 therapy effectively inhibited the growth of HCC in mice (A) After 4 weeks of plasmid injection in C57BL/6 J mice, a primary hepatocellular carcinoma model was established and the mice were randomly divided into four groups (Placebo group, n = 7; Anti-PD1 group, n = 7; FAK inhibitor group, n = 7; Combination group, n = 8), and specific information about the administration (time, dosage, and method). (B) The mouse liver after 2 weeks of medication. (C–D) The liver weight of mice and the liver weight/body weight of mice were compared in each group (Placebo group, n = 7; Anti-PD1 group, n = 7; FAK inhibitor group, n = 7; Combination group, n = 8). Significance identification: ns, p ≥ 0.05; *, p < 0.05; **, p < 0.01; ***, p < 0.001. (E) HCC tissues of mice were histologically analyzed by H and E staining (scale bars, 400 μm).

    Article Snippet: 2) anti-PD1 group: 200 μg PD1 antibody (anti-mPD1 clone RMP1-14, BioXcell, Cat. # BE0146) was injected by intraperitoneal injection, once every 3 days; 3) FAK inhibitor (VS4718) group: 50 mg/kg FAK inhibitor (VS4718) (Csnpharm, Cat. # CSN16593) dissolved in 0.5% methylcellulose (v/v, saline) was given to mice by gavage, twice a day.

    Techniques: Inhibition, Plasmid Preparation, Injection, Staining

    Combination of FAK inhibition and anti-PD1 therapy inhibited proliferation and promoted apoptosis of HCC in mice (A) PCNA immunohistochemistry on HCC tissues of mice (scale bars, 100 μm). (B) HCC tissues of mice in each group were stained with TUNEL-staining (scale bars, 200 μm). (C) Quantification of PCNA staining ( n = 5 mice/group). (D) Quantification of TUNEL staining ( n = 3 mice/group). Significance identification: ns, p ≥ 0.05; *, p < 0.05; **, p < 0.01; ***, p < 0.001.

    Journal: Frontiers in Pharmacology

    Article Title: A FAK Inhibitor Boosts Anti-PD1 Immunotherapy in a Hepatocellular Carcinoma Mouse Model

    doi: 10.3389/fphar.2021.820446

    Figure Lengend Snippet: Combination of FAK inhibition and anti-PD1 therapy inhibited proliferation and promoted apoptosis of HCC in mice (A) PCNA immunohistochemistry on HCC tissues of mice (scale bars, 100 μm). (B) HCC tissues of mice in each group were stained with TUNEL-staining (scale bars, 200 μm). (C) Quantification of PCNA staining ( n = 5 mice/group). (D) Quantification of TUNEL staining ( n = 3 mice/group). Significance identification: ns, p ≥ 0.05; *, p < 0.05; **, p < 0.01; ***, p < 0.001.

    Article Snippet: 2) anti-PD1 group: 200 μg PD1 antibody (anti-mPD1 clone RMP1-14, BioXcell, Cat. # BE0146) was injected by intraperitoneal injection, once every 3 days; 3) FAK inhibitor (VS4718) group: 50 mg/kg FAK inhibitor (VS4718) (Csnpharm, Cat. # CSN16593) dissolved in 0.5% methylcellulose (v/v, saline) was given to mice by gavage, twice a day.

    Techniques: Inhibition, Immunohistochemistry, Staining, TUNEL Assay